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   https://doi.org/10.1016/j.jmps.2023.105449  

   Kumar, Aditya; Yavari, Arash (December 2023, Journal of the Mechanics and Physics of Solids)
2. Coacervation-Induced Remodeling of Nanovesicles

   https://doi.org/10.1021/acs.jpclett.3c00705  

   Mondal, Sayantan; Cui, Qiang (May 2023, The Journal of Physical Chemistry Letters)
3. Investigating Osteocytic Perilacunar/Canalicular Remodeling

   https://doi.org/10.1007/s11914-019-00514-0  

   Yee, Cristal S.; Schurman, Charles A.; White, Carter R.; Alliston, Tamara (August 2019, Current Osteoporosis Reports)

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4. COP1 controls light-dependent chromatin remodeling

   https://doi.org/10.1073/pnas.2312853121  

   Wang, Wenli; Kim, Junghyun; Martinez, Teresa S; Huq, Enamul; Sung, Sibum (February 2024, Proceedings of the National Academy of Sciences)

   Light is a crucial environmental factor that impacts various aspects of plant development. Phytochromes, as light sensors, regulate myriads of downstream genes to mediate developmental reprogramming in response to changes in environmental conditions. CONSTITUTIVELY PHOTOMORPHOGENIC 1 (COP1) is an E3 ligase for a number of substrates in light signaling, acting as a central repressor of photomorphogenesis. The interplay between phytochrome B (phyB) and COP1 forms an antagonistic regulatory module that triggers extensive gene expression reprogramming when exposed to light. Here, we uncover a role of COP1 in light-dependent chromatin remodeling through the regulation of VIL1 (VIN3-LIKE 1)/VERNALIZATION 5, a Polycomb protein. VIL1 directly interacts with phyB and regulates photomorphogenesis through the formation of repressive chromatin loops at downstream growth-promoting genes in response to light. Furthermore, we reveal that COP1 governs light-dependent formation of chromatin loop and limiting a repressive histone modification to fine-tune expressions of growth-promoting genes during photomorphogenesis through VIL1. 

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5. Retinal vascular remodeling in photoreceptor degenerative disease

   https://doi.org/10.1016/j.exer.2023.109566  

   Nguyen, Matthew; Sullivan, James; Shen, Wen (September 2023, Experimental Eye Research)

   Fliesler, S (Ed.)

   Abnormal vasculature in the retina, specifically tortuous vessels and capillary degeneration, is common in many of the most prevalent retinal degenerative diseases, currently affecting millions of people across the world. However, the formation and development of abnormal vasculature in the context of retinal degenerative diseases are still poorly understood. The FVB/N (rd1) and rd10 mice are well-studied animal models of retinal degenerative diseases, but how photoreceptor degeneration leads to vascular abnormality in the diseases remains to be elucidated. Here, we used advancements in confocal microscopy, immunohistochemistry, and image analysis software to systematically characterize the pathological vasculature in the FVB/N (rd1) and rd10 mice, known as a chronic, rapid and slower retinal degenerative model, respectively. We demonstrated that there was plexusspecific vascular degeneration in the retinal trilaminar vascular network paralleled to photoreceptor degeneration in the diseased retinas. We also quantitatively analyzed the vascular structural architecture in the wild-type and diseased retinas to provide valuable information on vascular remodeling in retinal degenerative disease 

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