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Title: Galaxy interactions are the dominant trigger for local type 2 quasars
ABSTRACT

The triggering mechanism for the most luminous, quasar-like active galactic nuclei (AGN) remains a source of debate, with some studies favouring triggering via galaxy mergers, but others finding little evidence to support this mechanism. Here, we present deep Isaac Newton Telescope/Wide Field Camera imaging observations of a complete sample of 48 optically selected type 2 quasars – the QSOFEED sample ($L_{\rm [O\, \small {III}]}\gt 10^{8.5}\, \mathrm{L}_{\odot }$; z < 0.14). Based on visual inspection by eight classifiers, we find clear evidence that galaxy interactions are the dominant triggering mechanism for quasar activity in the local universe, with 65$^{+6}_{-7}$ per cent of the type 2 quasar hosts showing morphological features consistent with galaxy mergers or encounters, compared with only 22$^{+5}_{-4}$ per cent of a stellar-mass- and redshift-matched comparison sample of non-AGN galaxies – a 5σ difference. The type 2 quasar hosts are a factor of 3.0$^{+0.5}_{-0.8}$ more likely to be morphologically disturbed than their matched non-AGN counterparts, similar to our previous results for powerful 3CR radio AGN of comparable [O iii] emission-line luminosity and redshift. In contrast to the idea that quasars are triggered at the peaks of galaxy mergers as the two nuclei coalesce, and only become visible post-coalescence, the majority of morphologically disturbed type 2 quasar sources in our sample are observed in the pre-coalescence phase (61$^{+8}_{-9}$ per cent). We argue that much of the apparent ambiguity that surrounds observational results in this field is a result of differences in the surface brightness depths of the observations, combined with the effects of cosmological surface brightness dimming.

 
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NSF-PAR ID:
10409254
Author(s) / Creator(s):
; ; ; ; ; ; ; ; ; ;
Publisher / Repository:
Oxford University Press
Date Published:
Journal Name:
Monthly Notices of the Royal Astronomical Society
Volume:
522
Issue:
2
ISSN:
0035-8711
Page Range / eLocation ID:
p. 1736-1751
Format(s):
Medium: X
Sponsoring Org:
National Science Foundation
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