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Recent discoveries of copious amounts of dust in quiescent galaxies (QGs) at high redshifts (z ≳ 1 − 2) challenge the conventional view that these objects have a negligible interstellar medium (ISM) in proportion to their stellar mass. We made use of theSIMBAhydrodynamic cosmological simulation to explore how dust and cold gas evolve in QGs and are linked to the quenching processes affecting them. We applied a novel method for tracking the changes in the ISM dust abundance across the evolutionary history of QGs identified at 0 < z ≲ 2 in both cluster and field environments. The QGs transition from a diversity of quenching pathways, both rapidly and slowly, and they exhibit a wide range of times that elapsed between the quenching event and cold gas removal (from ∼650 Myr to ∼8 Gyr). Contrary to some claims, we find that quenching modes attributed to the feedback from active galactic nuclei (AGNs) do not affect dust and cold gas within the same timescales. Remarkably, QGs may replenish their dust content in the quenched phase primarily due to internal processes and marginally by external factors such as minor mergers. Prolonged grain growth on gas-phase metals appears to be the key mechanism for dust re-formation, which is effective within ∼100 Myr after the quenching event and rapidly increases the dust-to-gas mass ratio in QGs above the standard values (δ_DGR ≳ 1/100). Consequently, despite heavily depleted cold gas reservoirs, roughly half of QGs maintain little evolution of their ISM dust with stellar age within the first 2 Gyr following the quenching. Overall, we predict that relatively dusty QGs (M_dust/M_⋆ ≳ 10⁻³ − 10⁻⁴) arise from both fast and slow quenchers, and they are prevalent in quenched systems of intermediate and low stellar masses (9 < log(M_⋆/M_⊙) < 10.5). This strong prediction poses an immediate quest for observational synergy between, for example, theJames WebbSpace Telescope (JWST) and the Atacama Large Millimetre Array (ALMA). 

Abstract The abundance and distribution of⁴⁴Ti tells us about the nature of the core-collapse supernovae explosions. There is a need to understand the nuclear reaction network creating and destroying⁴⁴Ti in order to use it as a probe for the explosive mechanism. The⁴⁴Ti(α, p)⁴⁷V reaction is a very important reaction and it controls the destruction of⁴⁴Ti. Difficulties with direct measurements have led to an attempt to study this reaction indirectly. Here, the first step of the indirect study which is the identification of levels of the compound nucleus⁴⁸Cr is presented. A 100-MeV proton beam was incident on a⁵⁰Cr target. States in⁴⁸Cr were populated in the⁵⁰Cr(p, t)⁴⁸Cr reaction. The tritons were momentum-analysed in the K600 Q2D magnetic spectrometer at iThemba LABS. 

Stony coral tissue loss disease (SCTLD) was first observed in St. Thomas, U.S. Virgin Islands (USVI) in January 2019. This disease affects at least 20 scleractinian coral species; however, it is not well understood how reef diversity affects its spread or its impacts on reef ecosystems. With a large number of susceptible species, SCTLD may not follow the diversity-disease hypothesis, which proposes that high species diversity is negatively correlated with disease prevalence. Instead, SCTLD may have a higher prevalence and a greater impact on reefs with higher coral diversity. To test this, in 2020 we resampled 54 sites around St. Thomas previously surveyed in 2017 or 2019 by the National Oceanic and Atmospheric Administration National Coral Reef Monitoring Program. These sites represented a variety of species diversity values [categorized into poor (<12 spp. rich.) and rich (12 spp. rich.)] in multiple disease zones (Endemic: disease present  9 months; Epidemic: disease present 2–6 months; Control and Emergent: disease present no disease/<2 months). We hypothesized that, contrary to the diversity-disease hypothesis, sites with high species diversity (as measured by species richness or Simpson’s index) would have higher disease prevalence within the epidemic zone, and that high species diversity sites would have a greater impact from disease within the endemic zone. Results indicated a significant positive relationship between disease prevalence and diversity in the epidemic zone, and a similar trend in the endemic zones. Additionally, a negative relationship was seen between pre-outbreak diversity and loss of diversity and coral cover within the endemic zone. This supports the hypothesis that higher diversity predicts greater disease impact and suggests that SCTLD does not follow the diversity-disease hypothesis. Within the epidemic zone, the species with the highest SCTLD prevalence were Dendrogyra cylindrus, Colpophyllia natans, and Meandrina meandrites, while in the endemic zone, Diploria labyrinthiformis, Pseudodiploria strigosa, Montastraea cavernosa, and Siderastrea siderea had the highest SCTLD prevalence. Understanding the relationship between species diversity and SCTLD will help managers predict the most vulnerable reefs, which should be prioritized within the USVI and greater Caribbean region.