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  1. Guangdong Province in Southeast China is noted for its numerous geothermal resources due to tectonic episodes, mainly occurred during the Cretaceous. The surface heat flow and geothermal gradient are the most direct ways to understand the temperature of the Earth. However, geothermal resources are poorly utilized in Guangdong Province due to limited numbers of boreholes and surficial hydrothermal fluids. To improve the understanding of underground temperature distribution in Guangdong Province, we have applied power-density spectral analysis to aeromagnetic anomaly data to calculate the depth of the Curie isothermal surface. Upward continuation is applied and tested to the magnetic data. The calculated Curie isotherm is between 18.5 km and 25 km below surface. The fluctuation in the depth range reflects lateral thermal perturbations in the Guangdong crust. In particular, the eastern, northern, western and coastline areas of the province have a relatively shallow Curie isotherm. By comparing the surface heat flow, geothermal gradient, distribution of Mesozoic granite-volcanic rocks, and natural hot springs, we conclude that during Mesozoic, magmatism exerted great influence on the deep thermal state of Guangdong Province. A shallow Curie isotherm surface, as well as numerous natural hot springs and high heat flow, show clear signatures of shallow heat sources. 
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  2. The long noncoding RNA COLDAIR is necessary for the repression of a floral repressor FLOWERING LOCUS C (FLC) during vernalization in Arabidopsis thaliana. The repression of FLC is mediated by increased enrichment of Polycomb Repressive Complex 2 (PRC2) and subsequent trimethylation of Histone H3 Lysine 27 (H3K27me3) at FLC chromatin. In this study we found that the association of COLDAIR with chromatin occurs only at the FLC locus and that the central region of the COLDAIR transcript is critical for this interaction. A modular motif in COLDAIR is responsible for the association with PRC2 in vitro, and the mutations within the motif that reduced the association of COLDAIR with PRC2 resulted in vernalization insensitivity. The vernalization insensitivity caused by mutant COLDAIR was rescued by the ectopic expression of the wild-type COLDAIR. Our study reveals the molecular framework in which COLDAIR lncRNA mediates the PRC2-mediated repression of FLC during vernalization. 
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