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  1. An EMS-based forward genetic screen was conducted in an apoptotic null background to identify genetic aberrations that contribute to regulation of cell growth in Drosophila melanogaster. The current work maps the genomic location of one of the identified mutants, L.3.2. Genetic crosses conducted through the Fly-CURE consortium determined that the gene locus for the L.3.2 mutation is p47 on chromosome 2R. 
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    Free, publicly-accessible full text available September 18, 2024
  2. Search for astrophysical electron antineutrinos in Super-Kamiokande with 0.01wt% gadolinium-loaded water. 
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    Free, publicly-accessible full text available July 10, 2024
  3. Free, publicly-accessible full text available June 1, 2024
  4. Cosmic-ray muons that enter the Super-Kamiokande detector cause hadronic showers due to spallation in water, producing neutrons and radioactive isotopes. These are a major background source for studies of MeV-scale neutrinos and searches for rare events. In 2020, gadolinium was introduced into the ultra-pure water in the Super-Kamiokande detector to improve the detection efficiency of neutrons. In this study, the cosmogenic neutron yield was measured using data acquired during the period after the gadolinium loading. The yield was found to be ð2.76  0.02ðstatÞ  0.19ðsystÞÞ × 10−4 μ−1 g−1 cm2 at an average muon energy 259 GeV at the Super-Kamiokande detector. 
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    Free, publicly-accessible full text available May 18, 2024
  5. We report a search for cosmic-ray boosted dark matter with protons using the 0.37 megaton years data collected at Super-Kamiokande experiment during the 1996–2018 period (SKI-IV phase). We searched for an excess of proton recoils above the atmospheric neutrino background from the vicinity of the Galactic Center. No such excess is observed, and limits are calculated for two reference models of dark matter with either a constant interaction cross section or through a scalar mediator. This is the first experimental search for boosted dark matter with hadrons using directional information. The results present the most stringent limits on cosmic-ray boosted dark matter and exclude the dark matter–nucleon elastic scattering cross section between 10−33 cm2 and 10−27cm2 for dark matter mass from 1 MeV=c2 to 300 MeV=c2. 
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  6. In this paper we share the seemingly ordinary community-building digital technologies that helped facilitate nine days of virtual professional development (PD) on the Electronic Textiles (hereafter e-textiles) unit for Exploring Computer Science (ECS). The e-textiles unit challenges teachers to learn new content about computing by designing functional circuitry in hands-on, personalized crafts, in ways that stimulate inclusive pedagogy and asset-based perspectives of students. Finding the right combination of supportive technologies spanned two years, including planning and two rounds of implementation (2020-2022), with careful reflection for re-design. We decided on a few seemingly basic digital technologies that supported the following design goals: 1) transparency of in-progress crafts, 2) community-building, and 3) connection to teachers’ everyday classroom practice. Below we share three technology choices that orient our revised PD model with explanations for those choices rooted in theory and practice. 
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  7. The mutation I.3.2 was previously identified in a FLP/FRT screen of chromosome 2R for conditional growth regulators. Here we report the phenotypic characterization and genetic mapping of I.3.2 by undergraduate students participating in Fly-CURE, a pedagogical program that teaches the science of genetics through a classroom research experience. We find that creation of I.3.2 cell clones in the developing eye-antennal imaginal disc causes a headless adult phenotype, suggestive of both autonomous and non-autonomous effects on cell growth or viability. We also identify the I.3.2 mutation as a loss-of-function allele of the gene centromere identifier (cid), which encodes centromere-specific histone H3 variant critical for chromosomal segregation. 
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  8. The mutation I.3.2 was previously identified in a FLP/FRT screen of chromosome 2R for conditional growth regulators. Here we report the phenotypic characterization and genetic mapping of I.3.2 by undergraduate students participating in Fly-CURE, a pedagogical program that teaches the science of genetics through a classroom research experience. We find that creation of I.3.2 cell clones in the developing eye-antennal imaginal disc causes a headless adult phenotype, suggestive of both autonomous and non-autonomous effects on cell growth or viability. We also identify the I.3.2 mutation as a loss-of-function allele of the gene centromere identifier (cid), which encodes centromere-specific histone H3 variant critical for chromosomal segregation. 
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