Shape Memory Alloy (SMA)-actuators are efficient, simple, and robust alternatives to conventional actuators when a small volume and/or large force and stroke are required. The analysis of their failure response is critical for their design in order to achieve optimum functionality and performance. Here, (i) the existing knowledge base on the fatigue and overload fracture response of SMAs under actuation loading is reviewed regarding the failure micromechanisms, empirical relations for actuation fatigue life prediction, experimental measurements of fracture toughness and fatigue crack growth rates, and numerical investigations of toughness properties and (ii) future developments required to expand the acquired knowledge, enhance the current understanding, and ultimately enable commercial applications of SMA-actuators are discussed.
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Deshielding effects on fatigue crack growth in shape memory alloys- A study on CuZnAl single-crystalline materials
The factors that affect the fatigue performance of shape memory alloys (SMAs), including fatigue crack growth (FCG) response, is far from being well-understood. In this study, we point to a mechanism that degrades the FCG performance considerably. We introduce the notion of FCG being affected by shielding and deshielding mechanisms, the former enhancing the resistance while the latter reducing the mate- rials’ resistance. We show that the deshielding mechanism creates additional driving forces (positive K contribution) of both Mode II and Mode I types (as much as 5e10 MPa m1/2) which accelerates the crack advance. The origin of the positive K component is associated with the localized martensite variant formation that is highly asymmetric with respect to the crack tip. We derive a resultant DK in excellent agreement with that measured based on experimental displacement measurements. Overall, this study represents an advancement of our understanding in FCG of SMAs by quantifying the deshielding mechanism.
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- Award ID(s):
- 1709515
- NSF-PAR ID:
- 10104825
- Date Published:
- Journal Name:
- Acta materialia
- Volume:
- 176
- ISSN:
- 1359-6454
- Page Range / eLocation ID:
- 155-166
- Format(s):
- Medium: X
- Sponsoring Org:
- National Science Foundation
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Abstract Catastrophic accidents caused by fatigue failures often occur in engineering structures. Thus, a fundamental understanding of cyclic-deformation and fatigue-failure mechanisms is critical for the development of fatigue-resistant structural materials. Here we report a high-entropy alloy with enhanced fatigue life by ductile-transformable multicomponent B2 precipitates. Its cyclic-deformation mechanisms are revealed by real-time in-situ neutron diffraction, transmission-electron microscopy, crystal-plasticity modeling, and Monte-Carlo simulations. Multiple cyclic-deformation mechanisms, including dislocation slips, precipitation strengthening, deformation twinning, and reversible martensitic phase transformation, are observed in the studied high-entropy alloy. Its improved fatigue performance at low strain amplitudes, i.e., the high fatigue-crack-initiation resistance, is attributed to the high elasticity, plastic deformability, and martensitic transformation of the B2-strengthening phase. This study shows that fatigue-resistant alloys can be developed by incorporating strengthening ductile-transformable multicomponent intermetallic phases.
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Polyurethane (PU) elastomers are among the most used rubberlike materials due to their combined merits, including high abrasion resistance, excellent mechanical properties, biocompatibility, and good processing performance. A PU elastomer exhibits pronounced hysteresis, leading to a high toughness on the order of 104 J/m2. However, toughness gained from hysteresis is ineffective to resist crack growth under cyclic load, causing a fatigue threshold below 100 J/m2. Here we report a fatigue-resistant PU fiber–matrix composite, using commercially available Spandex as the fibers and PU elastomer as the matrix. The Spandex fibers are stiff, strong, and stretchable. The matrix is soft, tough, and stretchable. We describe a pullout test to measure the adhesion toughness between the fiber and matrix. The test is highly reproducible, showing an adhesion toughness of 3170 J/m2. The composite shows a maximum stretchability of 6.0, a toughness of 16.7 kJ/m2, and a fatigue threshold of 3900 J/m2. When a composite with a precut crack is stretched, the soft matrix causes the crack tip to blunt greatly, which distributes high stress over a long segment of the Spandex fiber ahead the crack tip. This deconcentration of stress makes the composite resist the growth of cracks under monotonic and cyclic loads. The PU elastomer composites open doors for realistic applications of fatigue-resistant elastomersmore » « less
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ABSTRACT: Due to rock mass being commonly subjected to compressive or shear loading, the mode II fracture toughness is an important material parameter for rocks. Fracturing in rocks is governed by the behavior of a nonlinear region surrounding the crack tip called the fracture process zone (FPZ). However, the characteristics of mode II fracture are still determined based on the linear elastic fracture mechanics (LEFM), which assumes that a pure mode II loading results in a pure mode II fracture. In this study, the FPZ development in Barre granite specimens under mode II loading was investigated using the short beam compression (SBC) test. Additionally, the influence of lateral confinement on various characteristics of mode II fracture was studied. The experimental setup included the simultaneous monitoring of surface deformation using the two-dimensional digital image correlation technique (2D-DIC) to identify fracture mode and characterize the FPZ evolution in Barre granite specimens. The 2D-DIC analysis showed a dominant mixed-mode I/II fracture in the ligament between two notches, irrespective of confinement level on the SBC specimens. The influence of confinement on the SBC specimens was assessed by analyzing the evolution of crack displacement and changes in value of mode II fracture toughness. Larger levels of damage in confined specimens were observed prior to the failure than the unconfined specimens, indicating an increase in the fracture resistance and therefore mode II fracture toughness with the confining stress.
1. INTRODUCTION The fracturing in laboratory-scale rock specimens is often characterized by the deformation of the inelastic region surrounding the crack tips, also known as the fracture process zone (FPZ) (Backers et al., 2005; Ghamgosar and Erarslan, 2016). While the influence of the FPZ on mode I fracture in rocks has been extensively investigated, there are limited studies on FPZ development in rocks under pure mode II loading (Ji et al., 2016; Lin et al., 2020; Garg et al., 2021; Li et al., 2021).
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null (Ed.)Red blood cells (RBCs) are subjected to recurrent changes in shear stress and oxygen tension during blood circulation. The cyclic shear stress has been identified as an important factor that alone can weaken cell mechanical deformability. The effects of cyclic hypoxia on cellular biomechanics have yet to be fully investigated. As the oxygen affinity of hemoglobin plays a key role in the biological function and mechanical performance of RBCs, the repeated transitions of hemoglobin between its R (high oxygen tension) and T (low oxygen tension) states may impact their mechanical behavior. The present study focuses on developing a novel microfluidics-based assay for characterization of the effect of cyclic hypoxia on cell biomechanics. The capability of this assay is demonstrated by a longitudinal study of individual RBCs in health and sickle cell disease subjected to cyclic hypoxia conditions of various durations and levels of low oxygen tension. Viscoelastic properties of cell membranes are extracted from tensile stretching and relaxation processes of RBCs induced by the electrodeformation technique. Results demonstrate that cyclic hypoxia alone can significantly reduce cell deformability, similar to the fatigue damage accumulated through cyclic mechanical loading. RBCs affected by sickle cell disease are less deformable (significantly higher membrane shear modulus and viscosity) than normal RBCs. The fatigue resistance of sickle RBCs to the cyclic hypoxia challenge is significantly inferior to normal RBCs, and this trend is more significant in mature erythrocytes of sickle cells. When oxygen affinity of sickle hemoglobin is enhanced by anti-sickling drug treatment of 5-hydroxymethyl-2-furfural (5-HMF), sickle RBCs show ameliorated resistance to fatigue damage induced by cyclic hypoxia. These results illustrate that an important biophysical mechanism underlying RBC senescence in which cyclic hypoxia challenge alone can lead to mechanical degradation of the RBC membrane. We envision the application of this assay can be further extended to RBCs in other blood diseases and other types of cells.more » « less
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