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A proposed paradigm for out-of-equilibrium quantum systems is that an analog of quantum phase transitions exists between parameter regimes of qualitatively distinct time-dependent behavior. Here, we present evidence of such a transition between dynamical phases in a cold-atom quantum simulator of the collective Heisenberg model. Our simulator encodes spin in the hyperfine states of ultracold fermionic potassium. Atoms are pinned in a network of single-particle modes, whose spatial extent emulates the long-range interactions of traditional quantum magnets. We find that below a critical interaction strength, magnetization of an initially polarized fermionic gas decays quickly, while above the transition point, the magnetization becomes long-lived because of an energy gap that protects against dephasing by the inhomogeneous axial field. Our quantum simulation reveals a nonequilibrium transition predicted to exist but not yet directly observed in quenched s-wave superconductors. 

Oxytocin increases the salience of both positive and negative social contexts and it is thought that these diverse actions on behavior are mediated in part through circuit-specific action. This hypothesis is based primarily on manipulations of oxytocin receptor function, leaving open the question of whether different populations of oxytocin neurons mediate different effects on behavior. Here we inhibited oxytocin synthesis in a stress-sensitive population of oxytocin neurons specifically within the medioventral bed nucleus of the stria terminalis (BNSTmv). Oxytocin knockdown prevented social stress-induced increases in social vigilance and decreases in social approach. Viral tracing of BNSTmv oxytocin neurons revealed fibers in regions controlling defensive behaviors, including lateral hypothalamus, anterior hypothalamus, and anteromedial BNST (BNSTam). Oxytocin infusion into BNSTam in stress naïve mice increased social vigilance and reduced social approach. These results show that a population of extrahypothalamic oxytocin neurons plays a key role in controlling stress-induced social anxiety behaviors.