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1. Triad Constraints for Learning Causal Structure of Latent Variables

   Ruichu Cai, Feng Xie (December 2019, Advances in neural information processing systems)

   Learning causal structure from observational data has attracted much attention,and it is notoriously challenging to find the underlying structure in the presenceof confounders (hidden direct common causes of two variables). In this paper,by properly leveraging the non-Gaussianity of the data, we propose to estimatethe structure over latent variables with the so-called Triad constraints: we design a form of "pseudo-residual" from three variables, and show that when causal relations are linear and noise terms are non-Gaussian, the causal direction between the latent variables for the three observed variables is identifiable by checking a certain kind of independence relationship. In other words, the Triad constraints help us to locate latent confounders and determine the causal direction between them. This goes far beyond the Tetrad constraints and reveals more information about the underlying structure from non-Gaussian data. Finally, based on the Triad constraints, we develop a two-step algorithm to learn the causal structure corresponding to measurement models. Experimental results on both synthetic and real data demonstrate the effectiveness and reliability of our method. 

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2. High-dimensional causal discovery under non-Gaussianity

   https://doi.org/10.1093/biomet/asz055  

   Wang, Y Samuel; Drton, Mathias (October 2019, Biometrika)

   Summary We consider graphical models based on a recursive system of linear structural equations. This implies that there is an ordering, $$\sigma$$, of the variables such that each observed variable $$Y_v$$ is a linear function of a variable-specific error term and the other observed variables $$Y_u$$ with $$\sigma(u) < \sigma (v)$$. The causal relationships, i.e., which other variables the linear functions depend on, can be described using a directed graph. It has previously been shown that when the variable-specific error terms are non-Gaussian, the exact causal graph, as opposed to a Markov equivalence class, can be consistently estimated from observational data. We propose an algorithm that yields consistent estimates of the graph also in high-dimensional settings in which the number of variables may grow at a faster rate than the number of observations, but in which the underlying causal structure features suitable sparsity; specifically, the maximum in-degree of the graph is controlled. Our theoretical analysis is couched in the setting of log-concave error distributions. 

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3. Estimating Causal Effects from Learned Causal Networks

   Raichev, Anna; Tian, Jin; Ihler, Alexander; Dechter, Rina (October 2024, {IOS} Press)

   The standard approach to answering an identifiable causaleffect query (e.g., P(Y |do(X)) given a causal diagram and observational data is to first generate an estimand, or probabilistic expression over the observable variables, which is then evaluated using the observational data. In this paper, we propose an alternative paradigm for answering causal-effect queries over discrete observable variables. We propose to instead learn the causal Bayesian network and its confounding latent variables directly from the observational data. Then, efficient probabilistic graphical model (PGM) algorithms can be applied to the learned model to answer queries. Perhaps surprisingly, we show that this model completion learning approach can be more effective than estimand approaches, particularly for larger models in which the estimand expressions become computationally difficult. We illustrate our method’s potential using a benchmark collection of Bayesian networks and synthetically generated causal models 

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4. iSCAN: Identifying causal mechanism shifts among nonlinear additive noise models

   Chen, Tianyu; Bello, Kevin; Aragam, Bryon; Ravikumar, Pradeep (December 2023, Advances in Neural Information Processing Systems 36 (NeurIPS 2023) Main Conference Track)

   Structural causal models (SCMs) are widely used in various disciplines to represent causal relationships among variables in complex systems. Unfortunately, the true underlying directed acyclic graph (DAG) structure is often unknown, and determining it from observational or interventional data remains a challenging task. However, in many situations, the end goal is to identify changes (shifts) in causal mechanisms between related SCMs rather than recovering the entire underlying DAG structure. Examples include analyzing gene regulatory network structure changes between healthy and cancerous individuals or understanding variations in biological pathways under different cellular contexts. This paper focuses on identifying functional mechanism shifts in two or more related SCMs over the same set of variables -- without estimating the entire DAG structure of each SCM. Prior work under this setting assumed linear models with Gaussian noises; instead, in this work we assume that each SCM belongs to the more general class of nonlinear additive noise models (ANMs). A key contribution of this work is to show that the Jacobian of the score function for the mixture distribution allows for identification of shifts in general non-parametric functional mechanisms. Once the shifted variables are identified, we leverage recent work to estimate the structural differences, if any, for the shifted variables. Experiments on synthetic and real-world data are provided to showcase the applicability of this approach. 

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5. Condition number bounds for causal inference

   Gordon, S.L.; Kumar, V. M.; Schulman, L. J.; Srivastava, P. (January 2021, Proceedings of Machine Learning Research)

   de Campos, C.; Maathuis, M. H. (Ed.)

   An important achievement in the field of causal inference was a complete characterization of when a causal effect, in a system modeled by a causal graph, can be determined uniquely from purely observational data. The identification algorithms resulting from this work produce exact symbolic expressions for causal effects, in terms of the observational probabilities. More recent work has looked at the numerical properties of these expressions, in particular using the classical notion of the condition number. In its classical interpretation, the condition number quantifies the sensitivity of the output values of the expressions to small numerical perturbations in the input observational probabilities. In the context of causal identification, the condition number has also been shown to be related to the effect of certain kinds of uncertainties in the structure of the causal graphical model. In this paper, we first give an upper bound on the condition number for the interesting case of causal graphical models with small “confounded components”. We then develop a tight characterization of the condition number of any given causal identification problem. Finally, we use our tight characterization to give a specific example where the condition number can be much lower than that obtained via generic bounds on the condition number, and to show that even “equivalent” expressions for causal identification can behave very differently with respect to their numerical stability properties. 

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