More Like this

1. A 2.56-mm ² 718GOPS Configurable Spiking Convolutional Sparse Coding Accelerator in 40-nm CMOS

   https://doi.org/10.1109/JSSC.2018.2865457  

   Liu, Chester; Cho, Sung-Gun; Zhang, Zhengya (October 2018, IEEE Journal of Solid-State Circuits)
2. Magneto-structural Correlations in Ni ²⁺ –Halide···Halide–Ni ²⁺ Chains

   https://doi.org/10.1021/acs.inorgchem.1c02483  

   Blackmore, William J. A.; Curley, Samuel P. M.; Williams, Robert C.; Vaidya, Shroya; Singleton, John; Birnbaum, Serena; Ozarowski, Andrew; Schlueter, John A.; Chen, Yu-Sheng; Gillon, Beatrice; et al (December 2021, Inorganic Chemistry)
3. A 334 μW 0.158 mm ² ASIC for Post-Quantum Key-Encapsulation Mechanism Saber With Low-Latency Striding Toom–Cook Multiplication

   https://doi.org/10.1109/JSSC.2023.3253425  

   Ghosh, Archisman; Mera, Jose_Maria Bermudo; Karmakar, Angshuman; Das, Debayan; Ghosh, Santosh; Verbauwhede, Ingrid; Sen, Shreyas (March 2023, IEEE Journal of Solid-State Circuits)
4. Phosphorescence in Mn ⁴⁺ -Doped R ⁺ / R ²⁺ Germanates ( R ⁺ = Na ⁺ or K ⁺ , R ²⁺ = Sr ²⁺ )

   https://doi.org/10.1021/acs.inorgchem.2c01364  

   Novikov, Sergei A.; Lu, Yimin; Zhang, Weiguo; Halasyamani, P. Shiv; Hariyani, Shruti; Brgoch, Jakoah; Klepov, Vladislav V.; zur Loye, Hans-Conrad; Mozharivskyj, Yurij (June 2022, Inorganic Chemistry)
5. Ca ²⁺ release or Ca ²⁺ entry, that is the question: what governs Ca ²⁺ oscillations in pancreatic β cells?

   https://doi.org/10.1152/ajpendo.00030.2023  

   Fletcher, Patrick A.; Thompson, Ben; Liu, Chanté; Bertram, Richard; Satin, Leslie S.; Sherman, Arthur S. (June 2023, American Journal of Physiology-Endocrinology and Metabolism)

   The standard model for Ca 2+ oscillations in insulin-secreting pancreatic β cells centers on Ca 2+ entry through voltage-activated Ca 2+ channels. These work in combination with ATP-dependent K + channels, which are the bridge between the metabolic state of the cells and plasma membrane potential. This partnership underlies the ability of the β cells to secrete insulin appropriately on a minute-to-minute time scale to control whole body plasma glucose. Though this model, developed over more than 40 years through many cycles of experimentation and mathematical modeling, has been very successful, it has been challenged by a hypothesis that calcium-induced calcium release from the endoplasmic reticulum through ryanodine or inositol trisphosphate (IP3) receptors is instead the key driver of islet oscillations. We show here that the alternative model is in fact incompatible with a large body of established experimental data and that the new observations offered in support of it can be better explained by the standard model. 

   more » « less